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that's, stimulation of CD74 with exogenous MIF could result in an too much inflammatory condition, as MIF is capable of stimulating CXCR2 and CXCR4 receptors In combination with CD74. CXCR2 and CXCR4 receptors when activated promote influx of neutrophils and lymphocytes, respectively (63, 64). Fig. four: Diagram from the human MIF gene showing its